Risk of primary non-function after liver transplant is: {Ans: 4-6%}What can cause a post liver transplant patient whose initial postop neurological course is normal to develop sudden deterioration in neurological status? {Ans: Intracranial bleed}Which medication causes increased cholesterol levels? {Ans: Sirolimis(rapamycin/rapamune)}What is NAFLD? {Ans: Nonalcoholic Fatty Liver Disease- caused by obesity, hyperglycemia, elevated serum lipids, and high BP}Clinical presentation for hepatic artery stenosis(HAT) {Ans: Increase in LFTs due to decreased blood flow through the hapatic artery to new organ Delayed bile leak Persistent sepsis of unknown cause}What is NASH? {Ans: Non-alcoholic steatohepatitis. Can lead to cirrhosis 3rd common indication for liver TX.}Why are induction agents given pre- or intraoperatively in transplant? {Ans: Decrease incidence of acute rejection delay first rejection delay use of calcineurin inhibitors due to their nephrotixicity}Calcineurin inhibitors {Ans: Tacrolimus(prograf) Cyclosporine(neoral or gengraf)}Reason for not accepted for liver TX {Ans: Hepatocellular Carcinoma (HCC) outside Milan Criteria}Side effects of calcineurin inhibitors(tacrolimus and cyclosporine) {Ans: Tremors, headaches, HTN, nephrotoxicity, high glucose levels, hair growth or loss, increased risk of cancer}Antiproliferative agents {Ans: Azathioprine (imuran) Mycophenolate mofetil(cellcept) Mycophenolic Acid(myfortic)}S&S of rejection post liver TX {Ans: Fever, ictera(yellowing of sclera),