metabolic acidosis {Ans: E. increased acid production, loss of bicarb, diminished renal excretion of hydrogen C.M hyperventilation (compensatory), h/a, n/v/d, dehydration, hypotension pH <7.4 HCO3 <22}Hydrostatic pressure {Ans: Blood pressure- pushes fluid outside of the vessels, the force of fluid against the walls of a compartment- venous obstruction, Na and water retention}Antidiuretic hormone {Ans: Secreted by pituitary gland in response to water deficit, Na excess, or hypotension. Causes kidneys to reabsorb water, increasing plasma volume}Liquefactive Necrosis {Ans: nerve cells- brain- accumulation of pus}Adrenergic agents effect on K+ {Ans: albuterol, beta blockers, and alpha adrenergic antagonists cause K+ movement into the cell. Alpha adrenergic receptors shift K+ out of the cell}Renin Angiotensin Aldosterone System {Ans: Activated by low blood volume, triggers release of renin which converts angiotensinogen to angiotensin 1. ACE converts angiotensin 1 to angiotensin which causes arterial vasoconstriction and stimulates release of aldosterone. Aldosterone stimulates renal Na reabsorption and K+ excretion. Water is retained, less urine is produced, blood volume increases.}